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Department of Internal Medicine

Jay V. Solnick, M.D., Ph.D. View profile as PDF

Jay V. Solnick

Clinical Interests

Jay V. Solnick is an infectious diseases clinician and microbiologist who specializes in the treatment of a broad range of infectious diseases. His laboratory studies the molecular events in the pathogenesis of Helicobacter pylori using mouse and non-human primate models.




Infectious Diseases, Internal Medicine, Medical Microbiology and Immunology


Internal Medicine
Medical Microbiology and Immunology


Infectious Diseases
Medical Microbiology and Immunology

Center/Program Affiliation:


Center for Comparative Medicine
County Rd. 98 and Hutchison Dr.
Davis, CA 95616
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M.D., Columbia University, College of Physicians and Surgeons, New York, New York, 1986
Ph.D., University of North Carolina, Chapel Hill, North Carolina, 1977
B.A., UC San Diego, La Jolla, California, 1971


Stanford University, Palo Alto, California, 1986-87


Stanford University, Palo Alto, California, 1987-89


Pasteur Institute, Paris, 1993-1994
Stanford University, Palo Alto, California, 1989-94

Board Certifications:

American Board of Internal Medicine, 1989
American Board of Internal Medicine, Infectious Disease, 2010

Professional Memberships:

American Society of Microbiology
Fellow, Infectious Diseases Society of America

Select Honors and Awards:

B.A. with High Honors, University of San Diego, 1971

Select Recent Publications:

Fei YY, Schmidt A, Bylund G, Johansson DX, Henriksson S, Lebrilla C, Solnick JV, Boren T, Zhu XD. Use of Real-Time, Label-Free Analysis in Revealing Low-Affinity Binding to Blood Group Antigens by Helicobacter pylori. Anal Chem 2011 July 19. [Epub ahead of print]

Moore, ME, Boren, T, and Solnick, JV. Life at the margins: Modulation of attachment proteins in Helicobacter pylori. Gut Microbes, 2011, 2:42-6.

Perry, S, de Jong, BC, 2, Solnick, JV, Sanchez, ML, Yang, S, Lin, PL, Hansen, LM, Talat, N, Hill, PC, Hussain, R, Adegbola, RA, Flynn, J, Canfield, DR, & Parsonnet, J. Infection with Helicobacter pylori is associated with protection against tuberculosis. PLoS One, 2010, 5(1): e8804

Styer, CM, Hansen, LM, Cooke, CL, Gundersen, AM, Choi, SS, BErg, DE, Benghezal, M, Marshall, BJ, Peek, RM Jr, Boren, T, and Solnick, JV. Expression of the BabA adhesion during experimental infection with Helicobacter pylori. infect Immun, 2010, 78: 1593-600.

Raffatellu, M, George, M, Akiyama, Y, Hornsby, MJ, Nuccio, S-P, Paizao, TA, Butler, BP, Chu, H, Santos, RL, Berger, T, Mak, TW, Tsolis, RM, Bevins, CL, Dandekar, S, Solnick, JV, Baumler, A. Lipocalin-2 resistance of Salmonella enterica serotype Typhimurium confers an advantage during life in the inflamed intestine. Cell Host Microbe 2009, 5: 476-486.

Cooke, CL, An, HJ, Kim, J, Canfield, DR, Torres, J, Lebrilla, CB, and Solnick, JV. Modification of gastric mucin oligosaccharide expression in rhesus macaques after infection with Helicobacter pylori. Gastroenterol, 2009, 137: 1061-71.

Amundsen, SK, Fero, J, Hansen, LM, Cromie, GA, Solnick, JV, Smith, GR, and Salama, NR. Helicobacter pylori Addax helicase-nuclease and RecA protein promote recombination-based DNA repair and survival during stomach colonization. Mol Microbiol 2008, 69: 994-1007.